Tag Info

The frequency is individual, and known as tinnitus frequency or pitch. From Okamoto et al., 2010: Our target notched music introduced a functional deafferentation of auditory neurons corresponding to the eliminated frequency band, and because this frequency band overlapped the individual tinnitus frequency, the notched music no longer ...

Feeling as though you have seen a face before is perfectly normal. It may reflect actual similarities between the new face and the face you have seen before. There are people who genuinely look like each other, an example being celebrity look-a-likes. It may also reflect a commonly observed cultural/ethnic effect where people of a different ethnicity look ...

It is not widely thought that impaired function or destruction of the fusiform is sufficient to produce prosoganosia. It is currently widely held that face processing involves a network of regions in the occipital and temporal lobes (e.g., the occipital face area, posterior superior temporal sulus, anterior superior temporal sulus, anterior collateral ...

I believe the answer lies in minicolumnar morphology in the neocortex. It's been shown that the minicolumns of autsitics and gifted individuals have narrower minicolumns, with greater spacing between each minicolumn. It's speculated that this creates an increased ability to distinguish percepts. Here is a paper on the topic: Casanova MF, Switala AE, Trippe ...

This is only one possible pathway. There are many potential ones. Further the fact that this is possible does not mean that it is the case always. Stress activates the hypothalamic-pituitary-adrenal (HPA) axis. For example, a meta-analysis by Dickerson et al. (2004) demonstrated that an acute laboratory based stressor reliably increased cortisol levels, ...

It’s probably best to ask clinicians since papers usually don’t go into very specific details about patient interactions, but in general this is what I can conclude based on a bit of research. Neglect often co-occurs with other impairments as there can be a range of causes (such as stroke) arising from damage to various brain structures; so it should be ...

Yes, though not necessarily in the way that you are meaning - some prosopagnosics will have damage to the area in question, so you could say the activity is lower because there is not much left of it! Quick google reveals this paper: http://cercor.oxfordjournals.org/content/20/8/1878.abstract It's recent so will probably review the evidence you are ...

There are many, many more neuromodulators in the brain than that. Essentially anything that binds to G-protein coupled receptors could be considered a neuromodulator. Even "classic" fast neurotransmitters like GABA and glutamate have "neuromodulatory" receptors (GABA-B receptors for GABA, 3 different families of "metabotropic glutamate receptors"). ...

The best thing you can do is avoid nutritional deficiencies and exercise. In general, it's the same as for the rest of the body. Many people have subtle nutritional deficiencies that they may never know about their whole life just based on eating habits. As Chuck Sherrington said, neurotransmitter-based treatments are subject to homeostatic compensation, ...

I think this is an interesting, but difficult question to answer. Hemispatial neglect patients do not typically have problem with visual pathways per se. Wikipedia suggests that the disorder is most closely linked to damage of the temporo-parietal junction and posterior parietal cortex, areas associated with attention. Thus, the sensation (e.g. on the ...

I don't know much about this, but here are a few review articles (see here for more) that you could look at: Heilman, K. M., & Valenstein, E. (1979). Mechanisms underlying hemispatial neglect. Annals of Neurology, 5(2), 166-170. http://www.arts.uwaterloo.ca/~raha/CogSci600_web/Readings/Danckert3.pdf Parton, A., Malhotra, P., & Husain, M. (2004). ...