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I've been searching about this but all I found was this presentation, which is a bit too technical for me to understand, e.g: Visual Cortical Plasticity

In what way does neuroplasticity affect the visual cortex?

For example, a person that wears glasses, if he/she stopped using them completely or from time to time, will the visual perception and interpretation of blurred data get better by just training? Will that blur also be reduced with training?

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up vote 5 down vote accepted

Yes, the visual cortex is plastic, even in adults:

We measured adaptation in the responses of populations of cat V1 neurons to stimulus ensembles with markedly different statistics of stimulus orientation. We found that adaptation served two homeostatic goals. First, it maintained equality in the time-averaged responses across the population. Second, it maintained independence in selectivity across the population.

Plasticity is invoked for encoding information during perceptual learning, by internally representing the regularities of the visual environment, which is useful for facilitating intermediate-level vision--contour integration and surface segmentation. The same mechanisms have adaptive value for functional recovery after CNS damage, such as that associated with stroke or neurodegenerative disease. A common feature to plasticity in primary visual cortex (V1) is an association field that links contour elements across the visual field. The circuitry underlying the association field includes a plexus of long-range horizontal connections formed by cortical pyramidal cells. These connections undergo rapid and exuberant sprouting and pruning in response to removal of sensory input, which can account for the topographic reorganization following retinal lesions.

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One great example of such plasticity is the ability of early-blind people to utilize their V1 area to read Braille:

Functional relevance of cross-modal plasticity in blind humans (Cohen et al., 1997)

Excerpt from the abstract:

We conclude that blindness from an early age can cause the visual cortex to be recruited to a role in somatosensory processing.

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